The CDC and the Infectious Diseases Society of America released cautionary statements about a new swine influenza virus that has been increasingly detected in few inland states. The virus is a type A (H3N2) that contains the genetic footprint of a conventional influenza A virus, along with markers for H3 and most notably the M gene from the infamous 2009 influenza A (H1N1) pandemic virus.
With this mid-twentieth century title paraphrasing an interesting article by Petersdorf, Swarner and Garcia, I preface a question that has been bugging me: Why is it that glucose drops in (bacterial) CNS infections? While I’m not 100% sure of the answer, and the question keeps coming back, I feel I can venture some possible explanations:
I belong to the generation of doctors who didnt grow up with Haemophilus influenzae type b. However, the U.S. literature is still fresh with descriptions of Hib clinical manifestations. This is a nasty bug. I just cant imagine how overwhelmed we would be if Hib and methicillin-resistant Staphylococcus aureus hang out together in our communities.
Every year we fight new battles against influenza, an old yet rejuvenated virus. Antigenic variations, deviation from vaccine composition and antiviral resistance spice up our coexistence and force us to keep abreast. I propose you follow three quick steps before you enjoy some hot chocolate and a good movie in this cold winter.